In the aftermath of an acute attack, pupillary distortion may result if there is permanent sphincter damage and/or iris atrophy. Anterior lens capsule opacities, known as glaukomflecken, may result from damage to the anterior lens epithelium from high intraocular pressure. The angle may have permanent synechiae formation. Intraocular pressure may be low if the ciliary body is so ischemic that aqueous humor production is compromised. As the ciliary body recovers, normal aqueous humor production resumes and the intraocular pressure rises.